Saturday, June 30, 2012

Signs of Anemia | Reference and Education Bioethics

Anemia as hemoglobin below the normal plasma as a result of the decrease in the number of circulating red blood cells or hemoglobin abnormally low total content per unit volume of blood defined. Anemia by chronic blood loss, bone marrow disorders, increased haemolysis, infection, cancer, endocrine deficiency, kidney failure, and numerous other diseases are caused. Anemia can at least temporarily corrected with blood transfusions. In addition, anemia caused by a deficiency of nutrients such as iron, folic acid and vitamin B12 (cyanocobalamin) is essential for normal erythropoiesis.

Hemolytic anemia:
In the cell anemia is so fragile that they break easily as they go through the capillaries, especially by the spleen. Red blood cell life span is so short that the fragile damaged cells can be formed rapidly and can cause severe anemia. In hereditary spherocytosis, red blood cells are very small and round, instead of a double-concave disc. In the past, the pulp of the spleen and some other vascular bed was found, they are easy to break even by the compression slightly. Erythroblastosis fetalis in with Rh-positive fetal red cells by antibodies from the mother is Rh-negative attack. These antibodies are Rh-positive cells vulnerable to what will be a fast break and the child born with severe anemia. Very rapid formation of red blood cells in erythroblastosis fetalis cells were in an explosion, a large number of early forms of red blood cells induced from bone marrow are released into the blood are destroyed. In sickle cell anemia, which in 0 3: 1 0 percent of the West African and American blacks have called the cells of an abnormal form of hemoglobin, hemoglobin. Hemoglobin S is by point mutations in the gene for beta-chain, which causes a decrease in the solubility of the deoxy form. Sickel anemia is a disease homozygous recessive. Sickel anemia is characterized by a long life of pain episodes (crises), chronic anemia associated with hyperbilirubinemia and increased susceptibilty to infection usually begins in early childhood. when the hemoglobin is exposed to low concentrations of oxygen, it can be in a long crystals in the red blood cells. Elongated crystals, giving the appearance of sickle-cell anemia as a double-concave discs and occulate arteries.

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Source: http://www.artebioetica.org/2012/06/29/signs-of-anemia/

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